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Clarithromycin (Biaxin)

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Biaxin is a medication of macrolide antibiotics group. Biaxin fights bacteria in the body. Biaxin is also used together with other medicines to treat stomach ulcers caused by Helicobacter pylori.

Other names for this medication:
Abbotic, Adel, Aeroxina, Althromicin, Apo-clarix, Bacterfin, Biclar, Bicrolid, Binoclar, Biotclarcin, Bremon, Bremon unidia, Ciclinil, Cidoclar, Clabact, Clabel, Clacee, Clacina, Clacine, Clactirel, Clamycin, Clarimac, Clarimax, Clarimed, Clarimycin, Claripen, Clariston, Claritab, Claritron, Claritrox, Claritt, Clariva, Clariwin, Clarix, Clarocin, Clarogen, Claromac, Claromycin, Claron, Clarosip, Claryl, Clarytas, Clasine, Clathrocyn, Clatic, Claxid, Cleanomisin, Cleron, Clonocid, Clormicin, Derizic, Egelif, Eliben, Emimycin, Eracid, Euromicina, Ezumycin, Finasept, Fromilid, Geromycin, Gervaken, Glartin, Hecobac, Heliclar, Helimox, Helozym, Infex, Iset, Italclar, Kailasa, Kalecin, Kalixocin, Karid, Karin, Klabax, Klabet, Klabion, Klarifor, Klarigen, Klariger, Klarimac, Klarimax, Klarit, Klarith, Klarithran, Klarithrin, Klaritpharma, Klax, Klaz, Klazidem, Klerimed, Kleromicin, Klonacid, Kofron, Krobicin, Laricid, Larithro, Larizin, Laromin, Lekoklar, Likmoss, Lyoclar, Macladin, Maclar, Macrobid, Macrol, Macromicina, Mononaxy, Monozeclar, Naxy, Neo-clarosip, Neo-klar, Nexium hp7, Nutabact, Odycin, Onexid, Opeclacine, Orixal, Pre-clar, Preclar, Quedox, Rocin, Rodizim, Rolacin, Rolicytin, Synclar, Taclar, Uniklar, Veclam, Vikrol, Xylar, Zeclar, Zeclaren

Similar Products:
Cipro, Zitromax, Erythromycin, Azithromycin, Roxithromycin, Erythrocin, Zmax, Zithromax, Ery-Tab, Dificid, Erythrocin Stearate Filmtab, Eryc, EryPed, Erythrocin Lactobionate, Ilosone, PCE Dispertab


Also known as:  Biaxin.


Biaxin is used to treat many different types of bacterial infections affecting the skin and respiratory system. Biaxin is also used together with other medicines to treat stomach ulcers caused by Helicobacter pylori.

Biaxin fights bacteria in the body.

Biaxin is also known as Clarithromycin, Maclar, Klaricid, Klacid, Clarimac, Claribid.


Biaxin is available in tablets.

Take Biaxin orally.

Take Biaxin with full glass of water.

Take Biaxin with or without food.

Do not crush, chew, or break the Biaxin tablet. Swallow the pill whole.

Shake the Biaxin oral suspension well before measuring a dose. Measure the Biaxin oral suspension with a marked measuring spoon or medicine cup.

Take Biaxin for for 7 to 14 days.

The dosage and the kind of medication depend on the disease and its prescribed treatment.

Do not stop taking Biaxin suddenly.


If you overdose Biaxin and you don't feel good you should visit your doctor or health care provider immediately. Symptoms of Biaxin overdosage: nausea, vomiting, diarrhea, abdominal discomfort.


Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Protect from light. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Clarithromycin are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Do not take Biaxin if you are allergic to its components or to clarithromycin or to similar medicines such as azithromycin (Zithromax), dirithromycin (Dynabac), erythromycin (E.E.S., E-Mycin, Ery-Tab, Erythrocin), troleandomycin (Tao).

Do not take Biaxin if you're pregnant or you plan to have a baby, or you are a nursing mother.

Do not take Biaxin if you take astemizole (Hismanal), cisapride (Propulsid), ergot medicine such as ergotamine (Ergomar, Ergostat, Cafergot, Ercaf, Wigraine), or dihydroergotamine (D.H.E. 45, Migranal Nasal Spray), pimozide (Orap), terfenadine (Seldane).

Do not take Biaxin if you have liver disease, kidney disease, myasthenia gravis, porphyria; personal or family history of "Long QT syndrome".

Try to be careful with Biaxin usage in case you are taking any prescription or nonprescription medicine, herbal preparation, or dietary supplement.

Avoid consuming alcohol.

It can be dangerous to stop Biaxin taking suddenly.

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We investigated the susceptibility to antibacterials of streptococci isolated from 8 medical facilities in Gifu prefecture between 2005 and 2007. Strains used in this study include 118 of Group A streptococci, 89 of Group B streptococci, and 58 of group G streptococci. For Group A streptococci, cefteram and imipenem gave the lowest MIC90 at 0.0078 microg/mL, followed by cefditoren and cefcapene at 0.0156 microg/mL and penicillin G, amoxicillin and meropenem at 0.0313 microg/mL. For Group B streptococci, cefteram, cefditoren, cefcapene and imipenem gave the lowest MIC90 at 0.0313 microg/mL, followed by penicillin G and meropenem at 0.0625 microg/mL and amoxicillin at 0.125 microg/mL. For Group G streptococci, cefteram and imipenem gave the lowest MIC90 at 0.0078 microg/mL, followed by penicillin G, cefditoren, cefcapene and meropenem at 0.0156 microg/mL and amoxicillin at 0.0313 microg/mL. With Group A and B streptococci, the susceptibility data obtained in this study were compared with those of strains between 2000 and 2001. As for group A streptococci, the MIC50 and MIC90 of beta-lactam agents were about the same as those of previous study, however the MIC90 of quinolones increased about 2.5-fold and resistant strains were found. As for Group B streptococci, the MIC50 and MIC90 of almost all of the antibacterials were about the same as those of the previous study, however the MIC90 of clarithromycin increased about 10-fold, indicating that the resistant strains are widely spread.

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Patients presenting with tonsillopharyngitis, sinusitis or pneumonia were randomized to receive either of the test drugs provided they clinically qualified for empirical clarithromycin treatment. The study endpoints were clinical and bacteriological cure rates, tolerability and safety. The study was designed to test for non-inferiority with regard to cure rates.

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There is currently conflicting level 1 evidence in the use of long-term antibiotics for chronic rhinosinusitis without nasal polyps. The primary aim of this feasibility study was to optimise future randomised trial design by assessing recruitment and retention of patients alongside providing preliminary data on symptomatic control.

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To clarify the role of MMPs regulated by CAM in the progression of myocarditis. Design CAM was given to experimental rats with autoimmune myocarditis (EAM) from day -7 to day 21 (early treated group, n=6) or from day 1 to day 21 (late treated group, n=6) twice a day.

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Eradication of Helicobacter pylori cures and prevents the relapse of duodenal ulceration and also results in histological resolution of chronic active gastritis.

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Of 51 specimens, 41 (80.39%) showed positive results in bacteria culture. The antibiotic susceptibility was as follows: vancomycin-100%, moxifloxacin-100%, levofloxacin-92.31%, rifampin-90.91%, ciprofloxacin-81.58%, SMZ-TMP-67.65%, azithromycin-47.62%, clarithromycin-45.00%, ampicillin sodium and sulbactam sodium-35.90%, cefatriaxone-39.39%, cefuroxime sodium-30.43%, penicillin-8.33%.

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PCR studies have demonstrated evidence of Mycoplasma pneumoniae and Chlamydophila pneumoniae in the lower airways of patients with asthma.

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On an intention-to-treat basis. H. pylori infection was cured in 77% (95% CI: 65-88%) of patients. Minor side-effects including diarrhoea, nausea and taste disturbance were reported by 64% of patients. Ninety-five per cent of patients consumed > 95% of tablets. Metronidazole resistance was 29% but all cultures were sensitive to amoxycillin and clarithromycin.

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To test whether one week's triple therapy with omeprazole and two antibiotics is enough to induce healing of a peptic (gastric and/or duodenal) ulcer.

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Clarithromycin (CLM) has been known to increase the cyclosporine (CsA) trough level in human and feline organ transplant patients. However, the interaction of CLM with CsA has not been reported in dogs. In this study, the effects of multiple dosing of CLM on the pharmacokinetics of CsA in three healthy beagles were investigated. The treatments included CsA 10 mg/kg alone and CsA 10 mg/kg + multiple-dose of CLM 10 mg/kg. Co-administration of CLM with CsA resulted in significant increases of oral bioavailability of CsA. The results of our study suggest that administration of multiple therapeutic doses of CLM may decrease the required CsA dosage in CsA-based immunosuppressive therapy in renal transplanted dogs.

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To Buy Ciprofloxacin Online Uk compare eradication success rate of H. pylori treatment regimens with and without doxycycline.

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Helicobacter pylori, discovered 27 years ago, has remained the most prevalent infectious agent in the world. In the author's hypothesis, the increase of peptic ulcer prevalence in the 19-20th century could be attributable to the extended worldwide use of gastric tubes for secretory testing which led to the iatrogenic transmission of pathogenic strains. Helicobacter pylori outer membrane proteins (OMP), and duodenal ulcer promoting (dupA) proteins were identified as novel virulence factors, leading to the production of Buy Cefdinir 300mg pro-inflammatory cytokines, which could be future targets of therapy. There is no ideal first-line eradication of the infection and according to expert's opinion, the efficiency of these regimens has fallen gradually in recent years to unacceptably low levels; however, in the author's opinion this is a multifactorial phenomenon which can not be generalized. As alternative drugs, the efficiency of levofloxacin, furazolidone and rifabutin has been proven by meta-analyses. Sequential and bismuth-free quadruple therapies, although highly efficient, are not yet used on a large scale. The recurrence of the infection is 2.27%/year in developed and of 13.0%/year in developing countries. Spontaneous eradication occurred in 8-20% of the children and 5-11% of adults. The prevalence of clarithromycin resistance is increasing worldwide. In Hungary, it has reached 10.9% in county cities, according to a national survey. In a district of Budapest called Ferencváros, the prevalence between 2005 and 2009 was 16-22%, with no increasing trend. The development of enzymatic inhibitors (urease, carbonic anhydrase and gamma-glutamyl transpeptidase), modified antibiotics and efflux pump inhibitors seem promising ways because these compounds do not lead to resistance; however, none have yet been used in humans.

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Mean (± SE) RAUC Buy Clindamycin Online Australia values were: ketoconazole (15 studies, 131 subjects), 11.5 (±1.2); itraconazole (five studies, 48 subjects), 7.3 (±1.0); clarithromycin (five studies, 73 subjects), 6.5 (±10.9); and ritonavir (13 studies, 159 subjects), 14.5 (±2.0). Differences among inhibitors were significant (F = 5.31, P < 0.005). RAUC values were not significantly related to inhibitor dosage or to duration of inhibitor pre-exposure prior to administration of MDZ.

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To evaluate the clinical efficacy of H. pylori detection using a molecular test and treatment outcomes of the clarithromycin-based genotypic Buy Flagyl 2g Online resistance test.

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The effect of Helicobacter pylori eradication on reflux Buy Levofloxacin Online oesophagitis is unclear.

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As with other bacterial infections, successful treatment of Helicobacter pylori infections depends on the use of antibacterial agents to which the organism is susceptible. In this article, we use the proposed report card grading scheme (i.e. grade A, B, C, D, F) for the outcome of clinical trials, where intention-to-treat cure rates >95% = A, 90-95% = B, 85-89% = C, 81-84% = D and <81% = F. The goal of therapy is to consistently cure >95% of patients (e.g. provide grade A results). Like tuberculosis, H. pylori infections are difficult to cure and successful treatment generally requires the administration of several antibacterial agents simultaneously. Duration of therapy is also important and depends upon whether resistance is present; 14 days is often best. With few exceptions, worldwide increasing macrolide resistance now undermines the effectiveness of the legacy triple therapy (e.g. a proton pump inhibitor [PPI], clarithromycin and amoxicillin) and, in most areas, cure rates have declined to unacceptable levels (e.g. grade F). The development of sequential therapy was one response to this problem. Sequential therapy has repeatedly been shown in head-to-head studies to be superior to legacy triple therapy. Sequential therapy, as originally described, is the sequential administration of a dual therapy (a PPI plus amoxicillin) followed by a Bazzoli-type triple therapy (a PPI plus clarithromycin and tinidazole) and has been shown to be especially useful where there is clarithromycin resistance. However, the cure rates of the original sequential treatment are grade B and can probably be further improved by changes in dose, duration or administration, such as by continuing the amoxicillin into the triple therapy arm. The sequential approach may also be more complicated than necessary, based on the fact that the same four drugs have also been given concomitantly (at least nine publications with >700 patients) as a quadruple therapy with excellent success. This article discusses the approach to therapy in the modern era where antimicrobial resistance is an increasing problem and legacy triple therapy is no longer an acceptable initial choice. Methods to achieve acceptable eradication rates (e.g. grade A or B results) are discussed and, specifically, sequential therapy is considered both conceptually and practically. Suggestions are provided regarding how sequential therapy might be improved to become a grade A therapy as well as how to identify situations where it can be expected to yield unacceptable results. New uses for current drugs are discussed and suggestions for subsequent randomized comparisons to overcome phenotypic and genotypic resistance are given. We propose a change in focus from comparative studies (designed to prove that a new therapy is superior to a known inferior therapy) to Buy Clarithromycin 500mg demanding that efficacious therapies meet or exceed a pre-specified level of success (i.e. grade A or B result). To do so, coupled with less concern about the effect of recommendations on the pharmaceutical industry, should provide clinicians with much higher quality information, and improve the quality of medical care and recommendations regarding treatment. Ultimately, there is little or no justification for comparative testing that includes an arm with known unacceptably low results. H. pylori gastritis is an infectious disease and should be approached and treated as such.

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ME was administered for 8-20 weeks in 68 adults with chronic sinusitis cases. The effect was evaluated in each factor from radiographic findings (R0-R3 according to the severity of the images), nasal findings (N0: no polyp, N1: a single polyp and N2: multiple polyps), allergic factors (A0: no allergy, A1: nasal allergy, A2: bronchial asthma) and objective nasal symptoms. In addition, an effect after polypectomy and histological examination were assessed for N1 and N2 groups.

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Hospitalized patients were evaluated prospectively during 3 winter seasons. In a clinical routine setting microbiologic tests were performed, nasal secretions were tested for viruses, Chlamydia pneumoniae and Mycoplasma pneumoniae, and clinical parameters were documented.

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With markedly increased antibiotic resistance and unsatisfactory efficacies of common empiric eradication regimens in the mainland of China, tailored therapy may be the best choice to achieve good efficacy. This study compared the eradication rates, safety, and compliance of tailored therapy to those of triple therapy plus bismuth and concomitant therapy in the naïve patients with Helicobacter pylori infection.